Emergency treatment in orbital fractures is always indicated in the following situations:
These injuries require ophthalmological intervention.
A pressure increase in the periorbital region due to a retrobulbar hematoma can cause significant injury such as the creation of compartment syndrome with injury of the neurovascular structures and the possibility of vision loss.
Read more details about retrobulbar hemorrhage here.
This CT scan sagittal view shows a small retrobulbar hematoma.
Coronal view of the same retrobulbar hematoma.
Axial view of the same retrobulbar hematoma.
If a retrobulbar hematoma leads to a tense, proptotic globe with acute visual disturbances, emergency decompression should be initiated. This situation requires urgent decompression, such as cantholysis, removal of implanted reconstructive material, and/or evacuation of a loculated hematoma under general or local anesthesia.
Transcutaneous transseptal incisions help evacuate the hematoma and relieve excess periorbital pressure. Alternative methods such as transconjunctival pressure release, lateral canthotomy, and inferior cantholysis may also be considered according to patient condition.
An exception may be pulsatile exophthalmos which can be a sign of a carotid-cavernous sinus fistula. A fistula of this nature requires appropriate preoperative imaging and planning.
Severe emphysema might significantly raise intraorbital pressure. If this compromises visual function or endangers the orbital contents, orbital decompression must be considered.
Patients with signs of intraorbital emphysema are given antibiotics and decongestive nasal drops.
To avoid additional emphysema due to acute pressure rise, patients with sinus fractures in the periorbital region should not blow their nose. They should also be instructed to sneeze with an open mouth to minimize the increase of intranasal/intrasinus pressure.
Usually, there is no need for emergency treatment of orbital floor/medial wall fractures unless there is severe ongoing hemorrhage in the orbital cavity, the paranasal, or nasal cavity, or fractures which create muscle ischemia.
In younger patients there is a danger of necrosis of the entrapped rectus muscle due to the so-called "trapdoor" phenomenon. In such cases, immediate release of entrapped tissues is necessary.
More information on the trapdoor phenomenon can be found in the orbital reconstruction treatment for orbital floor fractures in the Surgery Reference pediatric trauma section.
Special attention should be given to the posterior third of the orbit, the superior orbital fissure, and the optic canal. Fractures and hematoma formation in these anatomical areas can be associated with superior orbital fissure syndrome and optic nerve injury.
In this image the axial CT scan in the plane of the optic nerve shows multiple fractures of the lateral orbital wall and the greater wing of the sphenoid of the right side. Compression or displacement (stretching) of the optic nerve may be produced.
Displaced fracture fragments in the posterior third of the orbit may create nerve compression.
In case of severe nasal, oral, or pharyngeal hemorrhage, the following options should be considered:
Reduction of the fractured, inferiorly displaced orbital roof fragment may be sufficient to obviate the need for internal fixation hardware.
The unique and complex anatomy of the orbit requires significant contouring of the implants to restore the proper anatomy.
Most cases require reconstruction of the orbital roof to support the reduced bone fragments and restore the shape of the orbit. The reason for this is that the bony walls are comminuted, or bone fragments are missing. Therefore, one is reconstructing missing bone rather than reducing bone fragments. This can be accomplished using various materials.
There is hardly any anatomic region in the human body as controversial as this one in terms of appropriate material for use in fracture repair:
Many surgeons recommend using materials that allow anatomical sculpting, which are radiopaque, inert, and won’t migrate.
The use of resorbable materials is not indicated. Use of resorbable materials may lead to secondary changes of the orbital contours over time, which is undesirable.
Modern imaging analysis offers a unique chance to quantitatively assess the surgical result and stability over time. This can provide valuable information for future recommendations.
In the case of an intra- or subcranial approach, fixation of the orbital roof might be accomplished from inside the anterior cranial fossa.
Orbital roof fractures differ from orbital floor and wall fractures due to the pulsating dura that lies above. The pulsating dura and associated micromovements can eventually resorb autogenous or absorbable materials.
Advantages include the following:
Disadvantages include the following:
Advantages include the following:
The illustration shows a calvarial bone graft stabilized by a titanium fixation plate.
Disadvantages include the following:
A combination of titanium mesh and porous polyethylene is radiopaque and more rigid than porous polyethylene of a similar thickness. Some surgeons also believe that there is less risk of having retained sharp barbs, which can lead to entrapment of soft tissues during placement. This implant is designed to guarantee retention of a created shape, something which cannot be achieved by regular polyethylene without the memory provided by the titanium.
Other advantages are:
A composite of porous polyethylene and titanium mesh has the following disadvantage:
Advantages include the following:
Disadvantages include the following:
The illustration shows a non-thermoplastic port delivery system (PDS) implant.
Fixation of orbital reconstruction material varies with the type and nature of the fracture.
Fixation of most materials in the orbital floor is achieved using one or more screws. The diameter depends on anatomical requirements but will typically vary between 1.0 and 1.5 mm. Alternatively, other screw types can be used.
Exposure of orbital roof fractures is performed most commonly via a coronal approach, via existing lacerations, and upper blepharoplasty incisions.
Once the orbital roof is exposed, periorbital dissection is meticulously performed.
Care should be taken at the superior-posterior medial aspect where the optic nerve and the superior oblique muscles can be found.
Additional information about orbital anatomy and dissection can be found in the links below:
Periorbital dissection is performed after exposure.
Wide exposure and retraction, meticulous hemostasis, and good illumination (headlights, illuminated retractors) are critical to performing this procedure.
A foil (or sheet of other material) may help with retraction and help to avoid a prolapse of soft tissues, improve visualization, and prevent entrapment of soft tissues during implant placement.
The illustration shows the insertion of a foil above the retractor.
The retractor is removed, placed above the foil, and the orbital soft tissues are adequately retracted.
Many devices have been used to facilitate retraction of the orbital contents, including malleable retractors, spoons, and special orbital retractors designed for the globe (as illustrated).
In some cases, the orbital roof is reduced, and the fracture segment may be stable. Fixation may not be necessary. In these cases, the patient should have a close clinical and radiographic follow-up.
A careful assessment of the defect size should be performed preoperatively with the CT scan and confirmed intraoperatively by measurement.
It is controversial whether or not to extend the anterior part of the mesh over the supraorbital rim because it can cause discomfort and alter the aesthetics of the orbit. Whatever method is chosen (fixation anterior or posterior to the rim), the upper eyelid function must not be compromised.
Firstly, the mesh is cut.
Then all sharp edges of the plate are trimmed off to protect the soft tissues (note the shape of the fan has only a minimum number of screw holes).
After that, the mesh is contoured to achieve the required shape and accommodate key anatomical structures (nasolacrimal duct, infraorbital nerve, and optic nerve).
It is advisable not to extend the implant further posterior than 5mm anterior to the optic canal entrance (if the posterior support bone of the orbit can be reached).
A sterile artificial skull allows proper anatomical contour of the implant.
Restoration of the normal anatomy can be achieved with meticulous shaping, positioning, and fixation of the implant. Care should be taken to ensure that no soft tissue is entrapped during implant placement.
DICOM data can be used to generate 3D virtual models of the patient’s orbit, and a CAD-CAM custom shaped implant can be fabricated.
Further information about Computer Assisted Surgery can be found here.
Additionally, navigation may serve for intraoperative control of implant or fragment position.
Modern 3D C-arm technology will further improve intraoperative quality control of implant positioning. Click here for more information.
Endoscopic visualization, either through the maxillary sinus or intraorbitally, may additionally confirm implant placement and lack of incarceration of periorbital soft tissue.
Orbital roof implants must always be stabilized. Fixation is provided by at least one screw. The screw size must be appropriate for the chosen mesh type.
After the insertion of an implant and before closure, it is imperative to perform a forced duction test and to examine the state of the pupil.
If bone is harvested from a donor site, eg, cranial vault (parietal area), iliac crest, mandible, rib, etc, there may be donor site morbidity.
Once the bone is shaped to restore the normal anatomy, it should be rigidly fixed with plates and screws to limit micromovements and possible resorption.
Patient vision is evaluated on awakening from anesthesia and then at regular intervals until hospital discharge.
A swinging flashlight test may serve to confirm pupillary response to light in the unconscious or non-cooperative patient; alternatively, an electrophysiological examination must be performed but this is dependent on the appropriate equipment (VEP).
Keeping the patient’s head in a raised position both preoperatively and postoperatively may significantly reduce edema and pain.
Nose blowing should be avoided for at least ten days following fracture repair to prevent orbital emphysema.
The use of the following perioperative medication is controversial. There is little evidence to make solid recommendations for postoperative care.
Postoperative examination by an ophthalmologist may be requested. The following signs and symptoms are usually evaluated:
Postoperative imaging must be performed within the first days after surgery. 3D imaging (CT, cone beam) is recommended to assess complex fracture reductions. An exception may be made for centers capable of intraoperative imaging.
Ice packs are effective in the short term to minimize edema.
Remove the sutures from the skin after approximately five days if non-resorbable sutures have been used.
Avoid sun exposure and tanning to skin incisions for several months.
Diet depends on the fracture pattern.
A soft diet can be taken as tolerated until adequate healing of the maxillary vestibular incision.
Clinical follow-up depends on the complexity of the surgery and whether the patient has any postoperative problems.
With patients that have fracture patterns that include periorbital trauma, issues to consider are the following:
Other issues to consider are:
Following orbital fractures, eye movement exercises should be considered.
Generally, implant removal is not necessary except in the event of infection or exposure.
The patient needs to be examined and reassessed regularly. Follow-up imaging at 3–6 months is helpful to ensure proper pneumatization of the sinuses (particularly, mucocele formation must be ruled out), sealing of the skull base, and stability of fragment position.
Travel in commercial airlines is permitted following orbital fractures. Commercial airlines pressurize their cabins.
Facial fractures may predispose to Eustachian tube dysfunction due to pharyngeal swelling. Forced air insufflation by holding the nose, closing the mouth and attempting expiration and the use of decongestants can relieve middle ear pressure and drum discomfort.
Mild pain on ascent or descent in airline travel may be noticed. Flying in unpressurized aircraft, such as military planes, should be avoided for a minimum of six weeks.
No scuba diving should be permitted for at least six weeks.